Whether you live in a house or apartment or just need to ensure that your house is free from EMFs, there are a number of ways you can reduce exposure. One of the easiest is to limit your electronic device use. It is also possible to use EMF block paint to stop EMF radiation from reaching your home. emf radiation blocker to protect your house against EMF radiation is to put up a RF shielding canopy. This is a fabric made of net which contains EMF shielding and is used to block EMFs from entering rooms. emf blocking clothing is to have your home fitted with a conductive enclosure. They are referred to as Faraday cages.
Numerous studies have demonstrated how the non-ionizing energy of RF produces antiproliferative effects in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is thought to result from the deregulation the cancer stem cell. This could explain the long-term effects observed in certain patients suffering from advanced HCC. But, the reason for AM EMF's impact on patients with cancer is not evident.
Effects of AM RF EMF on HCC tumour growth in vivo were studied in mice. The tumors were split into 3 groups. First, the group that was unaffected to RF EMF. The second group was exposed to RF EMF at a frequency that is similar to the one used by humans. Third group members were exposed to the RF EMF in HCC-specific frequencies. The impact of HCCMF on tumors was assessed against the effect of RCF. The results indicated that tumors treated by HCCMF had significant shrinkage. However, the tumors treated with RCF showed no evidence of shrinkage in the tumour.
The reason for tumor-specific AM RF EMF may be based on the fact that tumour cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effect in HCC cells is caused by CACNA1H, a protein which mediates tumour-specific Ca2+ influx. The results suggest that CACNA1H could have wider implications for treatment and diagnosis of many cancers.
The tumours in the control group were not exposed to RF EMF, and were fed a normal mouse diet. emf radiation blocker of the HCCMF group were treated with Huh7 cells at the time they were between five and seven weeks old. The tumors were then killed in cases of excessive burden.
The tumours in the three groups showed distinct growth curves. The tumors treated with HCCMF had a significant reduction in size of the tumor after 8 weeks. However, tumors that were treated using RCF showed no shrinkage. The difference was significant. The tumours treated with RCF were able to show necrosis, which is common in tumours exposed to RCF. The possibility is that this necrosis was caused by an absence of oxygen in the larger tumours.
In sum, the results suggest an AM-RF EMF exhibits anticancer activity in vitro and in vivo. A number of studies have proven it is true that AM RF EMF produces measurable reduction in tumours in HCC patients. There is a possibility that AM RF EMF triggers these effects due to CACNA1H, a protein involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may exert a sustained influence on the growth of HCC tumors in vivo.